Annotation Detail
Information
- Associated Genes
- ALK
- Associated Variants
- ALK RANBP2-ALK
- Associated Disease
- inflammatory myofibroblastic tumor
- Source Database
- CIViC Evidence
- Description
- ALK-rearrangements are seen in 50% of inflammatory myofibroblastic tumor (IMT) cases. A 44 year old man was diagnosed with IMT. He was treated with surgery and catheter placement for administration of cisplatin, doxyrubicin and mitomycin C. Break-apart FISH showed ALK rearrangement, and immunohistochemical staining was characteristic of RANBP2 rearrangement. After further chemotherapy along with maintenance imatinib, crizotinib was started at 200mg twice daily, and a maximal partial response was achieved 5 months later, with regrowth occurring 2 months later. After further surgery, crizotinib was restarted at 250mg twice daily and almost 2 years later the patient was in complete radiographic remission. Another patient with non-ALK rearranged IMT was administered crizotinib in this study but did not respond.
- Variant Origin
- somatic
- Variant Origin
- Somatic
- Evidence URL
- https://civic.genome.wustl.edu/links/evidence_items/1244
- Gene URL
- https://civic.genome.wustl.edu/links/genes/1
- Variant URL
- https://civic.genome.wustl.edu/links/variants/514
- Rating
- 4
- Evidence Type
- Predictive
- Disease
- Inflammatory Myofibroblastic Tumor
- Evidence Direction
- Supports
- Drug
- Crizotinib
- Evidence Level
- C
- Clinical Significance
- Sensitivity/Response
- Pubmed
- 20979472
Drugs
Drug Name | Sensitivity | Supported |
---|---|---|
Crizotinib | Sensitivity | true |